{{ 'Go back' | translate}}
Njus logo

Health care: This CEO Says Telehealth Can Help Increase Patient Care | Njus Great Britain

This CEO Says Telehealth Can Help Increase Patient Care

Health care Giving Compass

'Giving Compass’ Take: • CEO of Lou Silverman of Advanced ICU Care describes the benefits to patient care with tele-ICU and other telehealth services. • How can donors support more telehealth initiatives in hospitals in order to address issues of access to healthcare? • Read about how telehealth is helping student populations have access to healthcare.St.Louis-based telehealth company Advanced ICU Care was born of simple necessity: Two intensivists were asked to care for patients in two different intensive care units (ICUs) simultaneously—but those ICUs were on opposite sides of the city.Thirteen years later, those two St.Louis hospitals remain clients of that company, which has been working to spread the concept of the tele-ICU.And even just five years ago, CEO Lou Silverman said he was still starting prospective client meetings with, “What (the heck) is tele-ICU?” In 2019, telehealth is playing an increasingly vital role in healthcare, and Silverman said the delineation between in-person and virtual healthcare—even in the most critical of care settings—is disappearing.Silverman recently sat down with FierceHealthcare to talk about the breakdown of the wall between virtual and in-person care.FierceHealthcare: Why is tele-ICU so important to the future of healthcare?Lou Silverman: People are getting older, older people understandably use more ICU resources, and the supply of intensivists is and will remain flat.Therefore, the demand for critical care expertise far outstrips the supply.FH: How is tele-ICU impacting costs for providers and payers?LS: Historically, our clients recognize between a two-to-one and a six-to-one return on investment (ROI) from our tele-ICU partnerships.While there are many factors that go into a sophisticated ROI calculation, some of the primary benefits include shorter ICU stays, improved utilization of ICU beds, fewer patient transfers, lower infection rates, fewer ventilator days and decreased incidence of sepsis.There are also additional clinical impact areas, such as reduced ICU mortality and adherence to well-established clinical best practices, which are material benefits of the program.These benefits arise in areas in which patients, providers and payers have alignment of interests.Read the full article about telehealth and patient care by Jacqueline Renfrow at Healthcare News. . The post This CEO Says Telehealth Can Help Increase Patient Care appeared first on Giving Compass .'

Treating Teens’ Depression May Make Parents Happier, Too

Health care Giving Compass

'Giving Compass’ Take: • The Atlantic reports that mental health treatment can have ripple effects in families — specifically, when teens receive counseling and therapy, their parents may feel some benefits. • Studies like this one only reinforce how important it is to ramp up mental health outreach and resources for young people.How can nonprofits make sure teens struggling with mental health get the help they need? • Here’s  how teens themselves are redefining the conversation around depression . I spent a lot of time in therapy as a kid, for depression, among other things.On and off until I graduated high school, I’d “hang out” in the doctor’s office, playing Connect Four before begrudgingly consenting to more intense discussions.The effect of these sessions was undoubtedly helpful for me.But one thing my self-involved teen brain never considered was that the treatment could improve my parents’ mental health as well.Preliminary new research, presented at the annual convention of the American Psychological Association [recently], suggests that it did: When depressed teens go through some version of mental-health treatment, symptoms of depression in their parents lessen.The finding, based on a study of 325 American teens and their parents, points to what might seem obvious in hindsight: Happier kids make for happier parents.It builds upon earlier research showing how mental health can be relational, hinting that mental-health care benefits not just individuals and their family members, but their entire communities … Few studies, however, have looked at how a child might affect their parent’s own mental health.Kelsey Howard, a doctoral candidate at Northwestern University and a co-author of the new research, says she suspects that’s because most of the research done so far has been concerned primarily with the treatment methods themselves, not on the effects of treatment on people’s relationships.Read the full article about teens’ depression and their parents by Angela Lashbrook at The Atlantic. . The post Treating Teens’ Depression May Make Parents Happier, Too appeared first on Giving Compass .'

Scientists find the earliest roots of Parkinson's disease in the brain

Health care The Conversation

New study challenges traditional view of Parkinson's disease.
'An array of positron emission tomography or PET images. Yok_onepiece/Shutterstock About 100 people have a rare mutation in a gene called SNCA that puts them at almost certain risk of getting Parkinson’s disease. This makes them ideal subjects for studying the root causes of this debilitating condition. Most of these people live in the northern Peloponnese in Greece, and a handful live in Campania, Italy. We were lucky enough to have 14 of these people agree to travel to London so we could study their brains. More than 6m people , globally, have Parkinson’s disease; it is the second most common neurodegenerative disorder after Alzheimer disease. The symptoms, which worsen over time, include motor symptoms such as stiffness, slowness and shaking, as well as non-motor symptoms, such as memory problems. Researchers have been trying to find a reliable marker for the disease so that people at risk can be identified before the motor symptoms start. There are no cures for Parkinson’s disease, but symptoms are treated with drugs that restore a brain chemical called dopamine to normal levels. Dopamine has long been considered a prime culprit in Parkinson’s disease as low levels cause problems with movement. But another brain chemical called serotonin has also been implicated in the disease. But we didn’t know how early and to what extent changes in serotonin occur and if these changes are related to disease onset. To help answer this, we needed to study those Greek and Italian subjects with the SNCA gene mutation. People with a rare genetic mutation that causes Parkinson’s hail from the Peloponnese in Greece. Lara Irimeeva/Shutterstock Studying these gene carriers before they develop Parkinson’s disease is a unique opportunity to understand what comes first in the cascade of events that eventually leads to a diagnosis of Parkinson’s disease. This knowledge is critical so that we can develop sensitive markers to track the progression of the disease. People with the mutation tend to display symptoms of Parkinson’s disease in their 40s or 50s, so we wanted to study subjects in their 20s and 30s to see if there were any brain changes a decade or more before symptoms started. Seven of our volunteers, who kindly visited our lab for ten days of brain imaging and neurological tests, had no motor symptoms and seven had been diagnosed with Parkinson’s disease. We also examined 25 patients with sporadic Parkinson’s disease (Parkinson’s disease without a genetic cause) and 25 healthy volunteers. All participant had three brain scans: one to measure dopamine, one to measure serotonin, and another to study anatomical regions in the brain. We also carried out a series of clinical tests to investigate motor and non-motor symptoms. The volunteers wore an electronic device on their wrist for seven days to pick up any movements associated with Parkinson’s disease – movement that might be too subtle to be detected by a neurologist with the naked eye. These tests confirmed that the seven subjects with the gene mutation who had no motor symptoms were, indeed, Parkinson’s free. Early serotonin loss Comparing data from the different groups allowed us to measure the severity of dopamine and serotonin loss at different stages of the disease, from people without symptoms to people with a diagnosis. It also allowed us to compare changes seen in the gene carriers with changes seen in those with sporadic Parkinson’s disease. This helped us translate our findings in the gene carriers into the more common sporadic form of Parkinson’s disease. We discovered that gene carriers without symptoms had depleted serotonin, while their dopamine neurons appeared to remain intact. So the changes in the serotonin system that we identified are likely to start very early and precede the onset of motor symptoms by some years. Our study, published in Lancet Neurology , suggests that changes to the serotonin system come first, occurring many years before patients show symptoms. This important finding could lead to the development of new drugs to slow or even stop disease progression. Our findings also suggest that brain scans of the serotonin system could be used as a tool for screening and monitoring disease progression. But these scans are expensive, so we need more work to develop affordable technology. We also need more research into genetic forms of Parkinson’s which could further unlock the earliest changes underlying this awful disease. Heather Wilson receives research support from the CHDI Foundation. Professor Politis research is supported by Michael J Fox Foundation for Parkinson’s Research, Edmond and Lilly Safra Foundation, CHDI Foundation, Glaxo Wellcome R&D, Life Molecular Imaging, Invicro, Curium, Medical Research Council (UK), AVID radiopharmaceuticals, National Institute for Health Research, Alzheimer’s Research UK, and European Commission IMI2 fund.'